Using transmission electron microscopy, we observed changes in the cytoplasm and peripheral vesicles.
Using transmission electron microscopy we observed that the diterpene induced a delay in the progression of cell division.
From a combination of confocal microscopy and transmission electron microscopy, we observed that the released pA disrupts the cell membrane.
By means of scanning electron microscopy, we observed indeed that the mutant parasites had a rougher, more heterogeneous surface, with apparently larger knobs, when compared to the wild-type parasites (Fig. 4B).
Using video-light microscopy, we observed the decrease in flagella beating frequency and severe changes in the lateral flange and in the general aspect of the cell.
Through further analysis via microplate reader, as well as confocal and wide-field epi-fluorescence microscopy, we observed non-specific binding of PI in viable filamentous cyanobacteria.
Using two-photon intravital imaging and transmission electron microscopy, we observed the existence of preferred sites for neutrophil entrance into the endothelial cell monolayer and exit from the basement membrane and pericyte sheath during neutrophil extravasation, namely, hotspots I and II, by elucidating distinctive roles of LFA-1 and Mac-1.
By confocal immunofluorescence microscopy, we observed that inner trabecular meshwork cells from anterior segments exposed to adenovirus (via injection into the inlet tubing during perfusion) had increased aquaporin-1 protein expression compared to endogenous levels.
By time lapse microscopy, we observed that SipA was injected earlier than SptP.
Through morphological analysis by fluorescence microscopy, we observed MC spreading in the presence of bFGF.
Using confocal microscopy we observed that NFAT5 was excluded from chromatin during mitosis in both isotonic and hypertonic conditions.
