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Following immuno-electron microscopy we detect SmAQP by immunogold labeling and observe gold particles often clustered in the parasite tegument.
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Using epifluorescence microscopy, we detected individual, freely moving filaments in solution in the absence of sAB-27 (Fig. 3a).
Performing confocal laser scanning microscopy, we detected E-cadherin in the plasma membrane of uninfected MCF-7 cells (Figure 4B and 4C, 'mock'mock
Using electron microscopy, we detected iron nanoparticles in the cytoplasmic compartment, enclosed within endosomes, suggesting that these particles are biologically inactive and cause little or no interference with cell physiology.
Using fluorescence microscopy we detected delivery of the YopK-βlac fusion protein into HeLa cells infected with a yopK null mutant (Figure 1E, upper right), but we did not detect delivery of the fusion protein into HeLa cells infected with the translocation-deficient yopB mutant (Figure 1E, upper left).
By light microscopy, we detected no apparent differences between E. bangladeshi and E. histolytica.
Using immunofluorescence microscopy, we detected reduced colocalization of TLR9 and the lysosomal marker Lamp-1 in UNC93B1-Y539A-expressing cells.
Using the cell-phasor approach to lifetime microscopy, we detected EGF-dependent FRET between EGFR eGFP and Grb2 mRFP.
In the human cell system, using confocal microscopy, we detected CCR2 expression being coexpressed in the elongated CD45+ cells.
By confocal microscopy, we detected a qualitative decrease in the amount of tubulin accumulation in myocytes using an anti-tubulin antibody that detects all forms of the protein.
In the present study, by using immunofluorescence microscopy, we detected EGFR-positive CTCs in 38%and44%4% of patients with early and metastatic disease, respectively, harboring CTCs in their blood.
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