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Using confocal laser scanning microscopy, we confirmed that the OsSND2-GFP fusion protein was located predominantly in the nucleus (Fig. 2a).
Using high-resolution X-ray diffraction and scanning transmission electron microscopy, we confirmed that the film was epitaxially grown without any significant lattice mismatches.
Using these unique patient populations and immune-electron microscopy, we confirmed that CD154 was an alpha granule and not a cell surface protein, and thereafter optimized the methods for its in vivo measurement in humans.
Using transmission electron microscopy, we confirmed that activated PBLs were eliminated mainly by necrotis (Figure 1).
Using electron microscopy, we confirmed that GABA-immunopositive axons made symmetric synapses with the cell body and the dendrites of mutant Purkinje cells (Fig. 2B).
Through live-cell microscopy, we confirmed that green fluorescent protein (GFP -tagged versions of these misfoldinGFP -taggedteins formed intracellular aggregates in immortalized rat striatal progenitor cells (ST14A) with similar characteristics as widely documented in the literature [23]–[26].
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By combining optical and Atomic Force microscopy we confirm the selective adsorption of gold nanoparticles, dendrimers and bacteria.
With the ability to visualize actin dynamics using fluorescence confocal microscopy, we confirm that TCR clusters and actin centripetal retrograde flow concurrently move inward to the center of the synapse.
Using live cell FRET analyses, biochemistry, subcellular fractionation and cryo-immunoelectron microscopy we confirm that only the GTP-restricted form, Sar1H79G, allows segregation of Cx43 from the ER membrane into ER exit sites (ERES).
Using secondary ion mass spectroscopy and transmission electron microscopy (TEM), we confirmed that the implanted Fe atoms diffuse out of the SiO2 layer and form Fe particles on both the SiO2 surface and the interface between SiO2 and Si.
Using transmission electron microscopy (TEM), we confirmed that there was indeed an increase in the number of autophagosomes/autolysosomes with accumulated substrates in the lumen (Fig. 4G).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com