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Light and electron microscopy revealed a disorganization of mitochondria and a dramatic decrease in mitochondrial density, even revealing regions completely lacking mitochondria in the KO hearts.
Electron microscopy revealed a good preservation of the fine structure.
Scanning electron microscopy revealed a smooth glassy surface.
Cryo-transmission electron microscopy revealed a PLGA core coated with one or several concentric lipid bilayers.
The live cell microscopy revealed a plausible explanations for the high mast cell death and putative outgrowth mechanism of C. albicans.
Transmission electron microscopy revealed a good state of CNT dispersion in the polymeric matrix.
The percolation parameters in conjunction with the optical microscopy revealed a kinetic rather than statistic percolation.
X-ray diffraction combined with transmission electron microscopy revealed a mixed nanocomposite morphology.
Scanning electron microscopy revealed a micro-porous structure, resulting from PEGDA gelation aligned with the bundles of CNF.
Evaluation of mitochondrial morphology with confocal and electron microscopy revealed a similar induction of fragmentation following global brain ischemia.
Super-resolution microscopy revealed a 80-nanometer distance between vertices, as well as unsuspected gaps in the mesh – weak points that may allow the red blood cell to reshape itself without breaking.
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