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Electron microscopy of glomerulus from transgenic mice showed extensive podocyte foot process effacement.
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Electron microscopy of one glomerulus showed diffuse foot process effacement, endothelial cell swelling and some loops with flocculent material between the endothelial cell and the glomerular basement membrane.
The second sequence shows time-lapse intravital microscopy of a glomerulus depicting glomerular capillaries in the superficial renal cortex of a Cx3cr1 gfp/+ mouse.
Electron microscopy of a single glomerulus showed numerous fibrils with a diameter of 10.6 13.8 nm and within an expanded mesangial matrix.
Immunoelectron microscopy revealed that immunolabeling of α-actinin 4 is disturbed and increased in the foot process of podocytes of glomerulus and in the microvilli of proximal tubules.
Kidney microscopy showed glomerulus congestion, intra-parenchymal congestion and tubular degeneration in phosphide poisoning (Mehrpour et al. 2008).
There was no change in histopathology of glomerulus and tubules of kidney.
Staining was markedly enhanced in the majority of glomerulus, mesangial cells, and tubular interstitium of untreated GS rats.
Accordingly, EM analysis of kidneys from these mice revealed normal configuration of glomerulus (Fig 3Bi).
The polygon selection mode was used to draw a line over the area of glomerulus.
Podocalyxin is a sialoprotein critical to maintaining integrity of filtration function of glomerulus.
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