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b Fluorescence microscopy imaging shows significant intensity of red fluorescence (580/605 nm) indicating the presence of intracellular red fluorescent microspheres.

Atomic force microscopy imaging shows significant and reversible changes of pore diameter at the extracellular mouth of Cx26 hemichannels exposed to different [Ca2+]e [Ca2+]e

Atomic force microscopy imaging shows significant and reversible changes of pore diameter at the extracellular mouth of Cx26 hemichannels exposed to different [Ca2+]e, however, the underlying molecular mechanisms are not fully elucidated.

(b,c) Live microscopy imaging showing adhesion of VarO-infected erythrocytes stained with PKH67 (green) to uninfected erythrocytes (b) or K562 cells (c) stained with PKH26 (red).

Atomic force microscopy and scanning electron microscopy imaging show the morphological structure of the multilayer film modified electrode.

Confocal laser scanning microscopy imaging showed that the depth of the bacterial biofilm on the anode was about 65 μm.

(b) Live microscopy imaging showing non-specific adhesion of MACS-purified NF54 GIE (upper panel) and uninfected erythrocytes (lower panel) stained with PKH67 (green) to human primary erythroblasts or K562 cells stained with PKH26 (red).

Cell uptake measurements were done using fluorescent signal analysis, FACS and microscopy imaging, showing enhanced abilities of SPMs to penetrate cells and tissues.

Atomic force microscopy imaging showed that the attached organic layer is thick while cyclic voltammetry experiments confirmed that this modified surface behaves as an ultramicroelectrode array.

Consistent with these findings, fluorescence microscopy imaging showed that both HIV-1 Gag and MLV Gag exhibited reduced puncta formation and an increased localization in large vacuoles in MiR+ cells as compared with Ctrl cells (Fig. 4B and 4C).

TEM findings as well as data obtained from the confocal microscopy imaging showed many living bacterial cells in the macrophages (Fig. 3D).