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DIC analysis and transmission electron microscopy demonstrate that slip occurs in a highly localized manner.
Immunofluorescence light microscopy and immunoelectron microscopy demonstrate that 3A protein, expressed in the absence of other viral proteins, colocalizes with membranes derived from the ER.
The results obtained from confocal microscopy demonstrate that chitosan dextran sulfate nanoparticles and cells are fully encapsulated within alginate microparticles, and spatially dispersed in the microparticle matrix.
Atomic force microscopy and confocal fluorescence microscopy demonstrate that approximately 30% of diamond nanocrystals with a size of less than 10 nm are fluorescent and have a remarkably long spin decoherence time (2.7 μs for a 7 nm diamond nanocrystal).
Biophysical studies (gel filtration, SDS PAGE, dynamic light scattering, thioflavin T assay, and electron microscopy) demonstrate that in contrast to wild type Aβ these targeted mutations lose the ability to self-associate.
Scanning Electron Microscopy and Scanning Electrochemical Microscopy demonstrate that the dispersion covers the whole surface of the electrode although there are areas with higher density of CNT and, consequently, with higher electrochemical reactivity.
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Confocal microscopy demonstrated that Gal-3 co-localized with the cryptococcal capsule (Fig. 6g and Supplementary Figure 2).
Scanning electron microscopy demonstrated that a lower seeding density results in a denser tissue.
Scanning electron microscopy demonstrated that the cells exhibited a normal morphology.
Confocal microscopy demonstrated that the collagen fibers were thin and arranged in a wispy pattern in E16 fetal rat wounds and in nonwounded dermis.
Scanning electron microscopy demonstrated that biofilms were present on the surface of five of five control catheters but only one of five treated catheters (P =.048).
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