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In addition, transmission electron microscopy analyses of the near-tip region have been performed to characterize cyclic deformation mechanisms.
X-ray diffraction and transmission electron microscopy analyses of the platinum carbon aerogel catalysts indicate the formation of well-dispersed platinum nanoparticles having sizes of about 3 nm.
SEM and light microscopy analyses of the dar1 SAM reveal an abnormal partitioning of meristematic domains, and mutations known to affect the SAM structure and function were found to interact with dar1.
The scratch failure mechanism studied through atomic force microscopy analyses of the scratches tracks is similar to the observed wear behavior of the dendrimer-mediated Cr and Cu films.
XRD and microscopy analyses of the olivine used in these experiments revealed the presence of magnetite (Fe3O4) and chromite (FeCr2O4) (Figures 1a and 1b), both of which are known to have a catalytic effect on the FTT reaction [23, 36 38].
Scanning tunneling microscopy and transmission electron microscopy analyses of deposited nanoparticulate films showed that the size and shape of synthesized nanoparticles can be changed dramatically from plate-like to aligned acicular when external magnetic field parallel to the plane of monolayer was applied during the synthesis of nanoparticles.
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As biomineral phases are soft materials, subjected to deterioration, amorphization processes, and artifacts formation [ 14], different preparation methods were used for electron microscopy analyses to monitor the reliability of the experimental results.
Western blot experiments and immunofluorescence microscopy analyses demonstrated transfer of recombinase-dependent rep68 genes into target cells.
The fluorescence microscopy analyses showed evidence of widespread iNOS expression in tumor cells from animals with MCA but no evidence of iNOS immunoreactivity in controls.
Similarly, immunofluorescence microscopy analyses revealed marked accumulation of both factors in nuclei of cells treated with fenretinide for 6 hours, compared to a diffuse distribution of NRF2 and HSF1 in untreated cells (Figure 4B).
Flow cytometry and time-lapse microscopy analyses confirmed that depletion of FBXO28 resulted in a progressive loss of proliferation, evident from around 36 48 h of siRNA silencing in several different tumour cell lines (Fig 1C and D; Supporting Information Fig S1A C).
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