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Although the surface of the eye appeared very irregular by scanning electron microscopy, microscopic sections confirmed the presence of relatively normal lenses, implying the presence of functional cone and pigment cells.
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Light and electron microscopic radial sections confirmed the near normal development of hair cells and supporting cells but also some degree of disorganization of both cell types (Fig. 6).
Microscopic analysis of brain sections confirmed the development of meningitis, showing inflammatory cell infiltration, hemorrhaging, thrombosis, edema and areas full of bacilli.
The microscopic examination of representative sections confirmed the radiological findings of infarct and showed generalized acute neuronal and glial necrosis, mild neutrophilic reaction and endothelial swelling in the left MCA, ACA and right ACA territories (Additional file 1: Figure S3).
Microscopic analysis of the brain sections confirmed that InhA is required for the development of meningitis.
The microscopic analysis of spinal cord sections confirmed the macroscopic regional distribution of DA receptors within the cord.
Fluorescence microscopic analysis of the tumour tissue sections confirmed expression of α5 β1 on the murine DU145 tumour model used.
A 3D reconstruction of confocal microscopic images, obtained from thick vibratome sections, confirmed that TLTs develop 3D networks of podoplanin+ cells wrapping around laminin+ matrix structures, similar to LNs.
Ex vivo fluorescence imaging of tumor sections and microscopic imaging confirmed significantly higher accumulation of the 2-DG dye in intracranial tumors than in normal brain.
Light microscopic sections were examined using a Leica DMRB brightfield microscope (objectives 1.6× to 20×).
The images are created after a specimen is embedded in gelatin, sliced into microscopic sections and digitized.
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