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Physico-chemical characterizations such as X-ray diffraction, laser Raman, and field emission scanning electron microscopic analyses revealed the formation of α-MnSe nanoparticles.
Microscopic analyses revealed that well-defined phase separated structures and uniform ionic pathway was formed for SPES-45-TPD membrane with the IEC of 2.29 mmol/g.
Immunofluorescence confocal microscopic analyses revealed that in HeLa cells containing endogenous Trak1, expression of GFP-tagged Trak1 WT, but not the GFP control, induced a mitochondrial hyperfusion phenotype with abnormally elongated and enlarged mitochondria (Fig. 4A C).
For the specimens tested at 760 °C/758 MPa microscopic analyses revealed that the thick specimens exhibited a mixed failure mode of void growth and cleavage-like fracture while the predominant failure mode for the thin specimens was cleavage-like fracture.
Western blotting and confocal microscopic analyses revealed that among the four 2As, the one derived from porcine teschovirus-1 (P2A) has the highest cleavage efficiency in all the contexts examined.
Fluorescence microscopic analyses revealed that the number of punctate LC3 structures -a hallmark of autophagy- was drastically higher in Tzb-refractory cells than in Tzb-sensitive SKBR3 parental cells.
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The microscopic analyses reveal the existence of thin coating of nitrogen-doped amorphous carbon on the surface of Co3V2O8 nanostructures.
Patterned films are obtained by photolithographic processes and microscopic analyses reveal that the resists provide a resolution of 4 μm and a good contrast behavior.
Electron microscopic analyses reveal that the platelike crystallites of the precursor are changed into nanowires with the diameter of ∼20 nm after the persulfate treatment.
However, confocal microscopic analyses reveal localization of VPS35 to intracellular punctate structures within dopaminergic neurons from rat primary midbrain cultures (Fig. 1C) or the intact rat substantia nigra (Fig. 1D), consistent with the localization of VPS35 to multiple vesicular compartments.
Electron-microscopic analyses reveal defects in collagen fiber density, diameter and arrangement as well as changes in the number and morphology of chondrocytic microvilli.
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