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The kinetics and thermodynamics of propionate degradation to methane, reversibility of the acetate oxidation process, and estimation of microbial growth are summarized.
Zero- and first-order degradation rates estimated at the laboratory scale by model fitting generally overpredict the rate of biodegradation when applied to the field scale because limited electron acceptor availability and microbial growth are not considered.
Considering the previous research suggestions on the fact that the levels of essential oils necessary to inhibit microbial growth are higher in foods than in culture media due the interactions between phenolic compounds and the food matrix, in our study we used the essential oils in the range of 500 and 2000 ppm [22].
A number of studies involving GFP-labeled strains of bacteria have revealed that GFP expression does not alter the biochemical, morphological, or survival characteristics of the labeled bacteria; however, only limited data regarding the effects of GFP expression on microbial growth are provided in these studies [11], [12].
In natural aquatic environments, nutrients required to support maximal microbial growth are rarely present in sufficient quantities in the water column.
To address this problem, we developed a systems-level approach to identify gene targets to prevent the biofilm formation of P. aeruginosa, and we found that genes essential to microbial growth are not necessarily good targets to treat P. aeruginosa biofilm, as most of the essential gene mutants form more biofilms before they are completely eliminated by antibiotics [ 9].
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Microbial growth is assumed to follow Monod-type kinetics.
Under laboratory conditions, microbial growth is maintained by using any P source, including Pt and HPt.
The current practice to control microbial growth is to include a biocide in the HF fluids.
Mixed-substrate microbial growth is of fundamental interest in microbiology and bioengineering.
Ruminal microbial growth was inhibited by 50% with 89 μg of ricin/mL.
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