Sentence examples for microbial exposure to from inspiring English sources

The very limited literature on microbial growth responses to CO and CO-RMs in vitro, and the transcriptomic and physiological consequences of microbial exposure to CO and CO-RMs are reviewed.

While low to high intensity pulsing can enhance productivities, increased microbial exposure to ultrasonic power causes cells to become fragile and reduces microbial viability due to mechanical stress on the microbial cell [ 92].

Expanding on previously published discussions (4 – 7 ), we outline the causal connections and reasons for their emergence, describing the breadth of the field and the diverse pathways from microbial exposure to chronic disease.

Bernstein et al. (2006) performed a controlled intervention in which ultraviolet radiation was applied in home ventilation ducts to reduce microbial exposures to fungally sensitized asthmatic children.

Previous studies have included direct assessments of oral (39) or gut (40) microbial exposures to study either cardiovascular or obesity risk; similar approaches can provide more precise characterizations of infection insulin resistance associations in future studies.

These data demonstrate the presence of a critical time window early in life during which appropriate microbial exposure has to occur in order to induce functional, life-long immune regulation that maintains serum IgE levels at baseline.

A key questions with regard to the hygiene hypothesis, which remains to be addressed, is how big the critical microbial exposure needs to be.

An infectious episode approach was designed in order to reduce the possibility of overestimation of microbial exposure due to multiple symptom and/or diagnosis reports during a single microbial infection.

The early work with the hygiene hypothesis was dominated by the conviction that Th1 and Th2 lymphocytes were mutually antagonistic, and that the decreased microbial exposure led to a reduced Th1 activity.

In circumstances of increased gut microbial exposure due to elevated gut epithelial permeability resulting from genetic deficiencies in local defense mechanisms or during gastrointestinal pathogenic infections, this paradigm is broken and a systemic immune response towards gut microbes is induced [ 20].

This study supports the concept that the intestinal epithelium has evolved to maintain a physiological state of inflammation with respect to continuous microbial exposure, which serves to sustain a tight intestinal barrier while preventing overt inflammatory responses that would compromise barrier function.