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The overall theory explaining microbial deterioration of concrete is quite well developed in the literature.
Microbial deterioration of polyurethane (PU) leather coating continues to represent a significant challenge in maintaining the durability and hygiene of leather products.
The cellulase enzyme complex was of keen military interest, it being a prime cause of the microbial deterioration of fibre and fabric: tents, cordage and clothing.
Initially assigned to assess microbial deterioration of Army materials, her studies crisscrossed fundamental study of the cellulase enzyme from diverse microbial strains, to enzyme structure, to synergism between hydrolase components, and to large-scale enzyme production.
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This makes lobster chitosan has a great potential to be used for preservation of foods from microbial deterioration.
According to these results, the enzymatically-responsive PU designed in this study has great potential as an ideal leather-finishing material, which is capable of providing efficient protection against microbial deterioration while minimizing undesirable side effects associated with biocide abuse.
We have previously shown that biosynthesized AgNPs can protect paint from microbial deterioration through antimicrobial activities [5, 10, 23], thus reiterating the relevance of AgNPs as antimicrobial additives in paint for applications in the built environment [58].
Exploitation of prooxidants as blended in polyethylene formation make it further susceptible to microbial deterioration.
Microbial grouting reinforcement to repair the deterioration of masonry structures involves injecting microorganisms and nutrient solution (a cementation solution) into existing granular system pores to induce the generated calcium carbonate cementation to form a microbial mortar of certain strength.
It is increasingly recognized that uraemic toxins originating from intestinal microbial metabolism may contribute to the deterioration of renal function, cardiovascular calcification, metabolic bone disease and mortality in CKD/HD patients [ 6, 7].
The transfer of a gene into the host from other microbial symbionts may have compensated for the deterioration of the gene in Buchnera, producing a selective advantage for the laterally transferred copy.
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