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This study helps us to understand microbial contributions to the etiology of Hirschsprung-associated enterocolitis.
This scheme illustrates the relative environmental and human microbial contributions to each sample based on a species-by-species literature survey based on species name searches in PubMed.
Further research on ecology of microorganisms in resource-limited tropical subsoils is warranted to better understand microbial contributions to biogeochemical cycles throughout tropical soil profiles.
Growing interest in microbial contributions to human health and disease has increasingly led investigators to examine the microbiome in both healthy skin and cutaneous disorders, including acne, psoriasis, and atopic dermatitis.
Microbial contributions to geomorphology have been particularly well studied in a number of extreme environments characterized by large swathes of bare rock, such as cold and hot arid environments, rocky coasts and coral reefs, and ruiniform landscapes.
The combination of detailed hydrological, biogeochemical, vegetation, and microbial data obtained from the BEO site2,10,52,53 suggest that under a warmer climate, microbial contributions to GHG emissions from these ecosystems will ultimately depend on how soil moisture (i.e., wetter vs drier6) is distributed across the landscape.
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Whilst recognition is increasing that life and landscapes are intimately related, as evidenced by growing research into ecosystem engineering, biogeomorphology and allied topics, the microbial contribution to such interactions has been relatively neglected.
Microbial contribution to duodenal non-ammonia N (NAN) flow was estimated by the PB/N ratio in bacterial fractions extracted from the liquid (LAB) and the solid (SAB) fraction of rumen digesta.
Transferring such high-octane bugs into lean mice causes the rodents to plump up, suggesting a microbial contribution to obesity.
Together these points support the microbial contribution to IBD, indicating that a basic antimicrobial mucosal barrier defect could be responsible for susceptibility to this disease.
While we are hopeful that results from this work enhance the understanding of the microbial contribution to NEC pathogenesis and suggest new approaches to targeted therapy, this study is not without limitations.
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microbial communities to
microbial cells to
microbial species to
microbial growths to
microbial proteins to
microbial responses to
microbial processes to
microbial contaminants to
microbial contribution to
microbial genomes to
microbial enzymes to
microbial strains to
microbial pathogens to
microbial populations to
microbial ecologists to
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