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The microarray was screened with the Zn150 and Zn2000 samples.
When the protein microarray was screened with the RR sera and compared to results from normal controls and unrelated autoantibodies, a number of targets of interest were identified (Table 2).
To test the hypothesis that fenretinide activates ER stress responses, a 24K microarray was screened for the induction of ER stress genes in SH-SY5Y neuroblastoma cells after a 6-h treatment with fenretinide.
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Finally, tumour tissue microarrays were screened for P-Akt and HER-2 expression.
The phage protein microarrays were screened to identify phage peptide clones that bind antibodies in serum samples from patients with sarcoidosis but not in those from controls.
When the GeneChip microarrays were screened with targets made from the SSH cDNA, 571 out of the 1409 transcripts were given "absent" calls, suggesting that no positive signals can be detected on the GeneChip microarrays for these transcripts, even though the presence of these 571 transcripts had been confirmed by sequencing.
Heteroplasmy was screened by microarray calculation with the method of Coon et al. [65].
Exosome was extracted from EGCG-treated 4T1 cells, and the change of miRNAs was screened using microarray.
The independent SAGE and microarray datasets were screened for S100 gene expression patterns.
In the current study, the selected transcripts from the microarray analysis were screened for differential expression by array-based quantitative PCR (QPCR).
The microarray data tables were screened for these 22 widely used reference genes, and 20 of them could be located on the Roche NimbleGen 12 × 135 K array that was used in our experiments.
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