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A GeneChip microarray study was designed to compare gene expression profiles of BZA to that of other SERMs, raloxifene (RAL) and lasofoxifene (LAS).
In order to gain insights into MPP+-induced neurotoxicity, a gene expression microarray study was performed using a midbrain-derived dopaminergic neuronal cell line, MN9D.
A microarray study was performed to identify the sdiA regulon of E. coli.
A microarray study was undertaken to define the gene expression profile in response to exosome treatment on a global scale.
Additionally, a second microarray study was performed recently that compared wild-type E. coli to sdiA mutant E. coli in late stationary phase at 30°C, although AHL was not included in the growth medium [34].
In order to obtain a global picture of gene expression changes associated with the activation of ALX receptor by the specific agonists AnxA1 and peptide Ac2-26, a whole-genome microarray study was carried out using GeneChip® Human Genome U133 Plus 2.0 (Affymetrix, Santa Clara, CA).
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Raw data of this microarray study are available at the GEO website (GSE2259).
It is worth noting that approximately half of the genes in our microarray study were down-regulated by Cobra1 knockdown.
Data from each microarray study were normalised by global normalisation.
Data from each microarray study were subjected to global normalisation.
The subjects of this microarray study were different from the current study.
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