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Aside from the technological issues, a difficult task in any microarray study is the analysis of the resulting enormous data set to reveal the key genes, whose regulation is central to the phenotypic changes observed.
Overall, the conservation of gene content among the MGEs of highly successful bovine SA isolates illustrated by the microarray study is consistent with evidence from prior studies and suggests a plug-and-play mechanism whereby a particular combination of MGEs enhances pathogenicity in a specific infection model [20].
Secondly, a genetic mutations identified by chromosome microarray study is not confined to advanced maternal age.
In addition, only half of the NPC samples express LINC00312, and the number of samples used in our microarray study is limited.
For example, suppose the goal of a microarray study is to identify genes differentially expressed with respect to an experimental treatment.
The physiological relevance of the genes identified by the microarray study is underscored by the finding that at least eight downregulated genes have already been shown to be essential for normal spermatogenesis.
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A GeneChip microarray study was designed to compare gene expression profiles of BZA to that of other SERMs, raloxifene (RAL) and lasofoxifene (LAS).
In order to gain insights into MPP+-induced neurotoxicity, a gene expression microarray study was performed using a midbrain-derived dopaminergic neuronal cell line, MN9D.
A microarray study was performed to identify the sdiA regulon of E. coli.
Raw data of this microarray study are available at the GEO website (GSE2259).
It is worth noting that approximately half of the genes in our microarray study were down-regulated by Cobra1 knockdown.
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