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In many microarray studies the primary objective is to identify, from a large panel of genes, those which are prognostic markers of a censored survival endpoint such as time to disease recurrence or death.
While UGT35b was consistently identified as rhythmic in all five microarray studies, the peak of expression in those studies occurred at ZT2 (±2 h).
For microarray studies, the ages of mice used were as follows: 6 weeks old (129S5 strain); 12 13 weeks old (C57BL/6j strain).
In parallel microarray studies, the B. anthracis Sterne ΔpXO1 strain affected 121 hBMEC genes by more than two-fold after 6 hours of infection compared to the uninfected control (Fig. 3C, Table S1B).
As was observed in our microarray studies, the transcript levels of IL-8, CXCL1 and CXCL2 were significantly downregulated in cells infected with B. anthracis Sterne compared to uninfected control or hBMEC infected with the ΔpXO1 strain (Fig. 5A).
Like other microarray studies, the primary objective of this study was to search for an optimal or near optimal subset of genes that could be used to predict the exposure history of unknown samples.
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Furthermore, from a transcriptome microarray study, the lncRNAs HIF1A-AS2 (HIF1A Antisense RNA 2) and AK124454 were shown to promote cell proliferation and invasion in TNBC (triple-negative breast cancers) cells and contribute to paclitaxel resistance13.
For each microarray study, the fold change and p-value of ING2 for the comparison were indicated.
From our microarray study, the most interesting candidates are the adjacent loci At2g23400 and At2g23410.
Strikingly, in the microarray study, the greatest-fold upregulated transcript was ofd1 itself.
In another miRNA microarray study, the role of miRNAs was investigated on gene expression of neutrophils after physical exercise.
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