Sentence examples for microarray studies show from inspiring English sources

Exact(5)

The results of the microarray studies show that the in vitro profiles of gene expression cluster per compound and incubation time, and when analyzed in a commercial gene expression database, can predict the toxicity and pathology observed in vivo.

Bioinformatics analyses based on EST sequences and exon-exon junction microarray studies show that 59% ∼74%of human genes are alternatively spliced [1], [2].

Microarray studies show that from 5-105-10%trarecripts arexpresseded with 24-hour rhythmicity.

Microarray studies show that transcripts for all of these genes, although not necessarily any untranslated regions, are present in ' Cand.

While the primitive streak is a region in which cells are losing epithelial characteristics, our microarray studies show that DLG1 expression is strongly upregulated in the primitive streak region relative to the epithelial epiblast.

Similar(55)

Microarray studies showed that gene expression spanning a region of approximately 84 kb upstream of the mutated gene was downregulated.

Microarray studies showed that subsets of genes induced by IFN-γ were inhibited by exosomes from H37Rv-infeced cells including genes involved in antigen presentation.

Indeed, recent microarray studies showed Nur77 induces known apoptotic genes such as FasL and TRAIL, but also novel genes such as NDG1 and NDG2 that promote apoptosis in a Bcl-2-independent manner [17].

In line with the pathogen dependent induction of AtWAKL10 and the ECGG50, microarray studies showed that the expression of the genes was induced by the functional synthetic SA analogue, benzothiadiazole S-methylester (BTH) (+1.59, 8 and 24 hat) in an NPR1-dependent manner (Figure 9A).

This finding corroborates previous microarray studies showing that, in general, more genes are upregulated than downregulated in aging liver [ 5].

Gene Ontology comparison of the present study with previous published murine microarray studies showed conserved Biological Processes during disease induction between the local joint and PBMC responses.

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