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Microarray studies of psychostimulant-induced changes in gene expression.
Several previous microarray studies of Li-induced changes in gene expression levels implied the involvement of inositol signaling-related genes.
Our modeling studies are complemented by experimental work being done at Princeton in the groups of Professors Josh Rabinowitz (mass spectroscopy) and David Botstein (microarray studies of gene expression).
We also use microarray studies of elongating hypocotyls to show that a number of growth-associated processes are activated by auxin including gibberellin biosynthesis, cell wall reorganization and biogenesis, and others.
It is also more comprehensive than prior microarray studies of SCZ.
Microarray studies of cells in which Oct4 is over/under-expressed [14], reveal an interesting result.
Microarray studies of SA isolates collected from humans also identified lineage-specific surface protein features [53].
Microarray studies of chronic hepatitis C infection have provided valuable information regarding the host response to viral infection.
We have previously found the KROX family transcription factor EGR1 to be differentially expressed in microarray studies of RNA extracted from whole scrapie-infected brains [4].
Previous microarray studies of CR have shown changes in Ddit4, Ghr [29] and Sgk1 [37] but not in most of these other genes.
Restricted sample size is one of common problems in microarray studies of difficult-to-obtain sample populations, such as female sporadic PD in our study.
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