Sentence examples for microarray series of from inspiring English sources

For this purpose, we performed hierarchical clustering in a previously published large microarray series of primary breast cancer patients (n = 850) with availability of distant metastases-free survival data (DMFS) [41].

The authors used receiver operating characteristic curves to determine the Ki-67 cut-off point that distinguished luminal-A from luminal-B tumors, then applied it to an independent microarray series of 4,046 breast cancers.

We downloaded Affymetrix microarray series GSE32426 consisting of rice embryogenic calli before and after Agrobacterium infection from NCBI GEO (http://www.ncbi.nlm.nih.gov/geo/) (Tie et al. 2012).

A microarray time series of PSM samples encompassing one period of the segmentation clock has been generated in the mouse and analyzed using the Lomb-Scargle (L) periodogram, a method related to Fourier analysis in that it attempts to fit the observed data to a sine curve [5], [11] (Microarray data are available at ArrayExpress at www.ebi.ec.uk/arrayexpress/ under accession number E-TABM-163).

Next, in a published microarray data series of 117 LUAD patients (GSE37138), we confirmed that the expression level of p27 negatively correlated with that of LUADT1.

Given the contradictory nature of our data, we therefore sought to further characterise S100A2 expression in an independent (from the microarray analysed) series of NSCLC patients, and to further investigate the role of S100A2 in early preneoplastic bronchial lesions.

A tissue microarray (TMA) series of breast cancer specimens from 1931 patients was immunostained for Ki-67, digitized with a whole slide scanner and uploaded to an image web server.

A tissue microarray (TMA) series of 596 hormone-naive prostate cancers (HNPCas) was screened for chromosome X and AR-gene locus-specific copy number alterations using four-colour fluorescence in situ hybridisation.

Given that tumor aggressiveness has been linked with EMT ([ 51], reviewed in [ 52]) we examined the expression of MYB in a publically available microarray dataset series of human breast tumors [ 50] and observed that MYB expression was generally high in Luminal A and Normal-like tumors but dramatically lower in Basal and Her2 + tumors.

By linking data from both ChIP-on-chip and expression microarrays, a series of differentially expressed genes regulated by changes of H3K9Ac and H3K9Me2 were identified.

Using oligonucleotide microarrays, a series of differential expression of genes was identified and further confirmed by RT-PCR.