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Microarray results suggested that OsIRO3 expression may be positively regulated by IDEF1 (Table 1).
An in silico analysis of our microarray results suggested that the canonical NF-κB pathway might be a pivotal effector of CO suppression in human monocytes.
Microarray results suggested that the expression level of Oct-4 self-renewal and stemness gene in LC-CD133+ was significantly up-regulated than that in LC-CD133−.
Previous microarray results suggested that miR-133b was significantly downregulated in GC [ 6].
Our microarray results suggested that podoplanin (PDPN) was up-regulated 13-fold in sPTK7 cells.
Microarray results suggested down-regulation of a C type MBL in HR.
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Relating these findings to our microarray results suggests that predicting classes in obesity may be improved by studying changes in gene expression once dietary intervention has begun (rather than single time points).
The global outcome of the IPA of microarray results suggests that CEsHUT mainly affected inflammatory and immune functions in human monocytes and that HDL opposed these functions due to its inhibitory effect.
Although it has been showed that the mutants in the BvrR/BvrS system have no obvious defects with regard to the ability to grow on standard media [4], our microarray results suggests that the BvrR/BvrS system controls elements directly involved in adjusting the Brucella metabolism to the nutrient shift expected to occur during the transit to the intracellular niche.
Thus, our microarray results suggest that a low dose of dietary resveratrol induces a transcriptional program similar to CR in multiple tissues and retards aging parameters, but these effects may be largely independent of the increase in SIRT1 activity and activation of Pgc-1α transcriptional targets reported previously for mice in a high fat diet fed high levels of resveratrol [8], [9].
Several genes from our microarray results suggest directions by which this hypothesis could be tested.
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