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Our microarray results do not show a significant correlation between ER status and OPG/RANKL expression.
However, the microarray results do not show any IFN mRNA targets to be affected by siQKI-tot.
In addition, the microarray results do not necessarily rule out the possibility that rare cellular subpopulations exist within primary tumors.
Functional analyses of the microarray results do not reveal a strong pattern of relationships among the set of 367 significant differentially expressed genes.
While these differences in gene signatures may offer a rational explanation for the symptoms, and confirm that CFS is a true and biological illness, gene microarray results do not imply that CFS is primarily a genetic disorder.
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Last but not least, the identification of the microarray results does not need any special instrument, whereas the ELISA method needs an enzyme immune analyzer, and other fluorescent immunological tests need an expensive fluorescent scanner or detector.
However, the microarray results did not show any change in the expression of the proU operon.
We analyzed three brain regions but the microarray results did not show gene expression differences between the tissues.
The microarray results did not show any altered expression of any known transcriptional regulator of AcrAB (acrR, marA, soxS neither ramA) (data not shown).
Interestingly, the microarray results did not show any significant increase in VvSTS expression in Corvina berries during both véraison and ripening.
Microarray results did not show cell cycle and toll-like receptor pathways as highly activated in HIV-1 activated macrophages, although the toll-like receptor pathway was highly enriched with known HIV-1 targeted proteins (Table 1).
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