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Glycan microarrays allowed identification of EV surface lectin(s), while plant lectin microarray profiling revealed terminal Man and GlcNAc residues exposed at the EVs surface.
Glycan microarrays revealed EV surface GlcNAc-binding lectin(s), while plant lectin microarray profiling revealed terminal Man and GlcNAc residues exposed at the EVs surface.
DNA microarray profiling revealed a lower expression of neuron-derived orphan receptor-1 (NOR-1) in elongated SMCs.
B-cell proliferation was measured by thymidine incorporation assay.Autoantigen microarray profiling revealed that pristane-treated IFNAR2-/ mice lacked autoantibodies directed against components of the RNA-associated autoantigen complexes Smith antigen/ribonucleoprotein (Sm/RNP) and ribosomal phosphoprotein P0 (RiboP).
Microarray profiling revealed distinct regulation of stress responses between xenograft tumors and parent cell lines, with gene ontology and network analyses linking gene expression to cell survival and metabolic processes.
On the other hand, microarray profiling revealed activation of multiple mucosal defence processes.
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Global microarray profiling reveals deficiencies in ECM gene expression in PKCα−/− skin correlating with abnormal collagen fibril morphology, disorganized dermal architecture, and reduced skin strength.
Thirdly, the M18 genome based microarray profiles revealed that the strain M18 has developed its specific temperature-dependent gene expression patterns to meet the requirements of survival and thriving in the rhizosphere niche.
Time-course microarray expression profiling revealed that normal and transformed cells transcriptionally responded to vorinostat treatment.
Microarray expression profiling revealed that 83 candidate genes are associated with Al stress and 25 are associated with tolerance.
Time-course microarray expression profiling revealed that normal and transformed cells transcriptionally responded to HDACi treatment, and that vorinostat signatures resembled other HDACi signatures in the Cmap database.
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