Sentence examples for microarray hybridization on from inspiring English sources

RNA labelling and microarray hybridization on wheat genome arrays were performed according to its manual.

We performed DASL assay and subsequent DNA microarray hybridization on the 25 paired FFPE samples.

For this reason we performed microarray hybridization on a subset of the 1,999 MPH-correlated genes.

Labelling and microarray hybridization on 385K RefSeq mouse promoter arrays were performed by Source BioScience imaGenes (Berlin, Germany).

The second step in this validation process was to perform gene-specific microarray hybridization on the multi-species cDNA microarray.

RNA samples were analyzed for their quantity and quality and further processed for the microarray hybridization on 12 × 135 K Roche NimbleGen arrays.

In total we performed 10 microarray hybridizations on males and 8 on females.

We have mapped SALL4 global gene targets using chromatin-immunoprecipitation followed by microarray hybridization (ChIP-on-chip) in myeloid leukemic NB4 cells [28], normal human CD34+ bone marrow cells, and 293 cells (unpublished data).

To determine the molecular mechanism(s) of SALL4 in normal hematopoiesis, leukemogenesis and ESC development, we have mapped SALL4 global gene targets using chromatin-immunoprecipitation followed by microarray hybridization (ChIP-on-chip) in normal human CD34+ bone marrow cells, myeloid leukemic NB4 cells and human ESCs.

We have mapped SALL4 global gene targets using chromatin-immunoprecipitation followed by microarray hybridization (ChIP-on-chip) in three types of cells that are related to SALL4 biological functions: leukemic cells [42], ESCs, and primary normal CD34+ cells (unpublished data), which have been correlated with the expression of these gene targets.

A published genome-wide profile of PPARα binding using chromatin immunoprecipitation combined with microarray hybridization (ChIP-on-chip) in HepG2 human hepatoma cells was then used to identify differentially expressed genes that had PPARα bound to their promoter regions.