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Microarray findings revealed the activation of gene groups that function in smooth muscle differentiation, signaling pathways, extracellular modeling and cell proliferation.
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This assumption has been recently challenged by our preliminary bioinformatics and microarray findings which revealed transcripts of aquaporin (AQP) 3 and 7 in human urothelium.
Bronchofiberscopic findings revealed no endobronchial abnormalities.
Pathological findings revealed a 35×34-cm34-cmmor.
Our findings revealed five key patterns.
The findings reveal other things, too.
The findings reveal 2 dynamic processes.
Consistent with these findings, microarray analysis revealed that the most robust changes in transcription were induced by Kdo2-Lipid A in wild type MEF cells, but that few changes were induced in ATF3-/ MEF cells.
However, in our study, neither microarray findings nor further analysis of real-time quantitative RT-PCR revealed any differences in level of transcription of the known virulence factors (CPS, suilysin, MRP and EF) between the wild type and mutant strain.
More recently two microarray studies revealed confirmatory findings with the downregulation of gamma-aminobutyric acid-associated receptors and the upregulation of glutamate-associated receptors in the kainite model of epileptogenesis as well as chronic human MTLE (3, 44).
This review summarizes the major microarray findings of relevance to neuropsychopharmacology, as a prelude to the design and analysis of future basic and clinical microarray experiments.
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