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Results obtained from these microarray experiments suggest that citrinin induced oxidative stress in the yeast cells.
DNA microarray experiments suggest the gene is not expressed above background levels, unlike C05D2.4/ bas-1 (Table 2).
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Our DNA microarray experiments suggested that transcriptions of ttha1934 and tthb071 were also up-regulated in ΔmutL, and ΔmutS2 cells, respectively (Supplementary Tables S3 and S4).
In contrast, our microarray experiments suggested about 11% of genes with overlapping antisense transcripts.
Taken together, these data confirm the results of microarray experiments suggesting that both miR-26a-1 and miR-16-2 are upregulated by p53 in response to doxorubicin.
The results of the microarray experiments suggested that expression of genes in the sterigmatocystin gene cluster was reduced in the xprGΔ1 mutant.
The small overlap between WRKY genes differentially expressed in response to jasmonate treatment in the microarray experiments suggested a tight developmental and/or spatiotemporal regulation in Arabidopsis.
However, we did not detect any IPT (involved in CK biosynthesis) or CKX (involved in CK degradation) genes as being significantly up- or down-regulated genes in our microarray experiments suggesting that the elevated endogenous CK concentrations previously reported may not be the result of increased or decreased activities of IPT and CKX genes, respectively.
It is noteworthy that NeuroD, a gene considered as directly regulated by Ngn proteins [ 19], was decreased in the Ngn2-/ embryos, but was not significantly upregulated after Ngn2 electroporation in microarray experiments, suggesting that the window for analysis of electroporated embryos was not suitable.
The candidate genes identified with the microarray experiment suggest the Clark genotype is capable of recognizing the iron deficiency and eliciting a change in transcription patterns as a response to the stress.
We have used the expression profiles from large-scale microarray experiments to suggest gene identities.
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