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These studies have helped illuminate some aspects of the microarray experiment; however, several fundamental observations of microarray behavior remain poorly understood.
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Microarray experiments, however, show that they may not bind to short probes and therefore may not be good targets.
Many transcripts were identified within the scope of the microarray experiments described, however only those associated directly with cuticle formation will be discussed here.
280 unique transcripts were identified within the scope of the microarray experiments described, however only those potentially associated with cuticle hardening will be discussed in this paper.
The identification of the regulative programmes from microarray experiments is limited, however, by the intrinsic difficulty of linear methods to detect low-variance signals and by the sensitivity of the different approaches.
However, in a microarray experiment, accurate assessment of the ratio of expression levels can be attained only if the fluorescent intensities can be accurately measured.
Transcription of other antioxidant genes (i.e., peroxidases, glutathione-S transferases, catalase) were indicated by our microarray experiment as affected by arsenate stress, however these genes were not included in our qRT-PCR validation.
However, this hypothesis relied on a single microarray experiment corresponding to only two time points in late S-phase.
However, often the sample size for each individual microarray experiment is small.
However, this gene was not differentially expressed in the microarray experiment.
However, since not all gene-results were validated by qPCR, results from this microarray experiment should be interpreted with care.
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