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Given that the GSEA of our microarray data suggests that SRA may negatively regulate TNFα signaling, we examined the ability of SRA to influence TNFα-induced JNK phosphorylation.
Congruence between testis histology and microarray data suggests that nearshore conch testes regress during the reproductive season, while offshore conch testes develop normally.
The microarray data suggests the presence of other transcriptional responses that may be linked to the sakacin P resistance phenotype particularly in L502-6.
By contrast microarray data suggests that END2 and PYS2 cells likely signal through the TAK1/p38/JNK and the TAK1/NLK pathways.
The fact that the mutant screen data which is more reliable than the microarray findings correlate well with the microarray data suggests that the above limitation is not a major concern.
Our microarray data suggests that alterations in elastic fibers and associated microfibrils, a major component of the ECM in the lamina cribrosa, may be a susceptibility factor predisposing the remodeling of the ONH in response to elevated IOP in glaucoma in the AA population.
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Microarray data suggested 65 immune related genes were commonly affected in the three treatment regimens.
The analysis and verification of proteome microarray data suggested that Pat acetylated several bacterial TFs.
Yet, the microarray data suggest that Esrrb and Mef2c are also rod-enriched (Fig. 1G, K).
These microarray data suggest that TPL2 expression in the prostate is inversely regulated by testosterone at the transcriptional level.
The microarray data suggest a decrease in transcript levels after day 15 for most of the genes studied.
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