In summary, the nCounter platform performed well, matching or exceeding Affymetrix microarray analysis in terms of sensitivity and precision using the same mass of total RNA as starting material.
The mRNA quantitation of 14 of these 18 genes closely mirrored the results of the microarray analysis in terms of up or downregulation and fold-change value, while the expression of the remaining four genes were corroborated in direction of change only.
For identification of differentially expressed genes (hereafter termed DEGs), microarray analysis was performed using the Partek genomic suite software version 6.3 (Partek, MO, USA) as previously described [ 30].
The results of our microarray analysis revealed that GO terms associated with "morphogenesis," "development" and "differentiation" were frequently decreased in the miR‐1‐positive breast carcinoma.
Microarray analysis data demonstrated that long-term potentiation (LTP) pathway was altered with down-regulation of genes against OS.
We performed a systematic search in the PubMed and EMBASE databases in May 2010 with the following medical subject headings (MeSH) terms: "meningioma", "microarray analysis", "oligonucleotide array sequence analysis", "gene expression profiling" (Table 1).
Microarray analysis from 8 biological replicates suggests that long-term sensitization training rapidly regulates at least 81 transcripts.
We selected 15 genes representing different functional categories, which according to our microarray analysis increased, decreased, or remained essentially unchanged in terms of transcript abundance following salt-shock.
To test whether pRb regulates cell adhesion-associated gene expression globally, we compared pRb-expressing and pRb-deficient osteoblasts in terms of gene expression patterns using microarray analysis.
Several functional terms that were not identified by microarray analysis were identified as enriched by 454 cDNA sequencing.
