Loeser, R.F. et al. Microarray analysis reveals age-related differences in gene expression during the development of osteoarthritis in mice.
Jackson, T. C., Kotermanski, S. E. & Kochanek, P. M. Whole-transcriptome microarray analysis reveals regulation of Rab4 by RBM5 in neurons.
Microarray analysis reveals that one mutant, p5325,26, is severely compromised for transactivation of most p53 target genes, and, moreover, p5325,26 cannot induce G1-arrest or apoptosis in response to acute DNA damage.
Microarray analysis reveals that AHK1/ATHK1 functions upstream of several stress-responsive transcription factors, such as AREB1, ANAC, and DREB2A, and is a positive regulator of drought and salt stress responses through both abscisic acid (ABA -dependent ABA -dependentndent signandng pathways [171].
HOTAIR also regulates BCSCs, and microarray analysis reveals HOTAIR overexpression upregualtes the genes related to stemness and EMT, such as CD44, STAT3, ALDH2, ZEB1 and VIM, but the tumor initiating frequency in vivo assay are needed to demonstrate the role of HOTAIR in regulating BCSCs further (Padua Alves et al., 2013).
In addition, microarray analysis reveals distinct differences between these cell lines.
DNA microarray analysis revealed that eight genes were downregulated in all comparisons (Tables 1, 2).
Microarray analysis revealed many novel genes differentially expressed by ventilation including matrix metalloproteinase-8 (MMP8) and GADD45α.
Mechanistically, microarray analysis revealed DEPTOR as a target of miR-375 in hASCs.
Our earlier DNA microarray analysis revealed expression of putative tumour suppressor exostosin 1 (EXT-1) in odontoblasts.
Microarray analysis revealed only 5 regulated genes in the 24-h group and 4 in the 72-h group.
