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Subsequent microarray analysis provided insight into the gene expression changes that result from 5-aza-2-deoxycytidine induced DNA demethylation.
Microarray analysis provided a detailed snapshot of the flux in the transcriptional network when Lbx1 is removed from the system.
The shared ATX1-AtMTM1 dehydration-responding fractidentifiedfied by microarray analysis provided further evidence for a pathway involving an epigenetic regulator and a lipid phosphatase producing PtdIns5P (microarray expression data are deposited in the NCBI series GSE15577).
Microarray analysis provided transcriptomic evidence for N- but not P-limitation in K. brevis.
Microarray analysis provided reproducible results over all biological and technical replicates.
The cDNA microarray analysis provided a global view of the dynamic transcriptome of wood formation.
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Microarray analysis provides the ability to obtain gene expression data on thousands of gene targets in parallel and is being applied to an increasing variety of neurobiological samples.
Overall, our microarray analysis provides further molecular support for this model.
Microarray analysis provides an opportune method for comparing, on a large scale, changes in gene expression that result from single factor manipulations.
The differentially expressed miRNAs detected by this microarray analysis provide a valuable candidate list of muscle growth and development related miRNAs.
Importantly, our microarray analysis provides a greater understanding of the patterns of activation of specific genes during endothelial differentiation, and identifies novel gene targets that may be used to enhance endothelial differentiation in the future.
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