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Chromosome choice was informed by microarray analysis implicating the chromosomes with the greatest expression profile change.
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Since the microarray analysis implicated an association between PT treatment and AM dysfunction, we investigated the role of these cells by depleting them before PT treatment and infection with influenza virus.
As OXY was shown to inhibit translation and microarray analysis implicated active autophagy, further evidence for induction of autophagy was sought.
Transcriptome profiling of genes regulated by p300/CBP has been analyzed by one study, using treatment with a p300/CBP inhibitor and microarray analysis, which implicated 615 as being regulated by p300/CBP.
The predictive protein interactome analysis implicated genes in addition to the ones revealed by the microarray studies, such as Drp1 and AKAP10.
A recent limited microarray-analysis also implicated possible ER stress signaling in PAC-1-induced cell death.
Novel data from cDNA microarray analysis and pathway inhibitor assays implicated several signaling pathways in OXY activity.
Intracellularly, YopM seems at least partly to be localized in the nucleus [16], [17], [18] and one study using microarray analysis found the dysregulation of several genes implicated in cellular growth and cell cycle control [19].
In contrast, microarray analysis identified expression changes in groups of genes implicated in oxidative stress, cell cycle regulation, and apoptosis, which is consistent with the chondrocyte pathology.
Now we have performed a microarray analysis in our mouse models to discover genes/transcripts potentially implicated in the pathogenesis of this CHD.
However, microarray analysis and real-time RT-PCR results from this study implicates their involvement in BLG or OVA sensitization.
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