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The CtrA regulon has been characterized by chIP-chip analysis, identifying genomic regions where CtrA binds, and microarray analysis, identifying genes with affected expression in a CtrA temperature sensitive mutant strain [31].
This combination of full genome microarray analysis identifying genes differentially expressed upon acquisition of doxorubicin resistance with an assessment of over-representation of doxorubicin pharmacokinetic or pharmacokinetic genes in the dataset provided significant insight into new pathways associated with doxorubicin resistance.
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Results: Microarray analysis identified that CHI3L1 is up-regulated specifically in inflamed mucosa.
Microarray analysis identified differential expression of 9 transcripts at a single age.
The microarray analysis identified 32 candidate genes that might be important for controlling the bulgy phenotype.
Microarray analysis identified single-copy losses of the region containing SATB2 in each of the individuals (Figure 1).
A recent study employing microarray analysis identified a set of miRNAs altered in spinal cord injury [49].
Second, microarray analysis identified a clear whole blood transcriptional signature that distinguished oAC-treated from untreated PbA-infected mice.
Microarray analysis identified a number of genes whose expression was altered in skeletal muscle of exercise-trained Myog-deleted mice.
Microarray analysis identified several growth factors and pathways that were differentially expressed in stroma in response to 3D epithelial culture.
A microarray analysis identified distinct gene expression patterns between control and tumors containing IRP2 or IRP1 transgenes.
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