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Achiron, A. et al. Microarray analysis identifies altered regulation of nuclear receptor family members in the pre-disease state of multiple sclerosis.
DNA microarray analysis identifies changed genes that are expressed at high or moderate levels.
Nevertheless, our microarray analysis identifies possible mechanisms underlying the protective effect of Alethea.
41 Chromosomal microarray analysis identifies many common causes of ASDs, such as the maternal duplication in chromosome band 15q11 q13; microdeletions and microduplications in 16p11.2, 22q11, and 22q13 deletion syndromes; Potocki-Lupski syndrome; and some cases with Angelman syndrome.
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Results: Microarray analysis identified that CHI3L1 is up-regulated specifically in inflamed mucosa.
Microarray analysis identified many miRNAs with increased or reduced levels during regeneration.
Expression microarray analysis identified ∼350 genes that were altered within 48 h of RA treatment.
MicroRNAs (miRNAs) may be responsible for variant OPN expression, interrupting translation by binding OPN messenger RNA (mRNA) in 3'-untranslated regions (UTRs).A microarray analysis identified miRNAs of interest.
Microcell-mediated chromosome transfer and expression microarray analysis identified nine genes associated with functional suppression of tumorogenicity in ovarian cancer cell lines; AIFM2, AKTIP, AXIN2, CASP5, FILIP1L, RBBP8, RGC32, RUVBL1 and STAG3.
Microarray analysis identified differential expression of 9 transcripts at a single age.
The microarray analysis identified 32 candidate genes that might be important for controlling the bulgy phenotype.
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