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Gene microarray analysis additionally identified two known MT1-MMP substrates known to be involved in growth and invasion, decorin and type I procollagen [ 28].
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Parallel microarray analysis has additionally identified a wide range of Pitx2 transcriptionally modulated left targets, possibly mediating its action.
These data confirm the results obtained with the microarray analysis and additionally show that the effects of TSA are time and stimulus dependent.
Additionally, microarray analysis indicated that TGF-β1 up-regulated MAPK signaling in contrast to C-ABC, which did not enrich genetic pathways.
Additionally, microarray analysis of these prototypical nephrotoxicants provided an opportunity for the development of candidate bridging biomarkers of nephrotoxicity.
Additionally, microarray analysis of wild type and Sin3A knockdown cells identified differences in the expression of several cell cycle control genes [ 9].
Additionally, microarray analysis allows for the discovery of new genes and/or pathways previously not known to be involved in a specific host-pathogen interaction.
Additionally, microarray analysis confirmed the strain-level synteny among S. Newport clusters and revealed subtle genomic differences within the clusters, showing that there were 0 to 30 gene differences among the S. Newport II isolates and 0 to 16 gene differences among the S. Newport III isolates (see Fig. S2 in the supplemental material).
Additionally, a microarray analysis was performed to examine a wider range of genes.
Additionally, the microarray analysis presented here, confirmed by the qRT-PCR and secreted cytokines analyses, showed that Src kinases inhibition had a similar effect on the transcriptional changes induced by TLR3 and TLR8 activation excluding a pathway-specific involvement of these kinases in the TRIF or MyD88 signaling cascades.
AW additionally carried out microarray analysis.
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