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DNA microarray analyses show that the evolved and ancestral viruses affect the global patterns of host gene expression in radically different ways.
Microarray analyses show that the absence of PKBα in early thymocyte subsets modifies the expression of genes known to be involved in pre-TCR signaling, in T cell activation, and in the transduction of interferon-mediated signals.
Coincident with this microvascular plasticity, results from microarray analyses show 9 out of 22 TGFβ-responsive mRNAs significantly up-regulated by 6 h post-ischaemia.
Microarray analyses show that adipose mass and depot differences in adipose tissue gene expression in mice are regulated by sexually dimorphic gene networks.
Microarray analyses show that transcript abundance of key glycosyl transferase genes known to be involved in wall synthesis or re-modelling did not match the increases in cellulose, GAX and lignin.
Our microarray analyses show that MAP2K3 may play a role in hypothalamic inflammation, and it also identified a gene, Hap1, that was up-regulated in response to expression of constitutively active MAP2K3.
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Furthermore, microarray analyses showed that the expression of OS-related genes in the mouse brain was also changed.
DNA microarray analyses showed that 396 genes (229 up-regulated and 167 down-regulated) were differentially expressed in the mutant MT-1 compared to WT.
Pathway specific microarray analyses showed that in addition to RANTES and IP-10, mycobacteria infected CFP10-DCs showed reduced expression of many Th1 promoting chemokines and chemokine receptors.
Real-time RT-PCR and microarray analyses showed significantly decreased Cyp17a1 expression (∼70%) in both siRNA1- and siRNA2-transfected MLTCs (Figure 2).
These observations were confirmed by microarray analyses showing that the gene expression profile characteristic of hESCs (high Oct-4, Nanog, Sox2, Cripto and Nodal, etc).
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