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Microarray analyses reveal that these increases trigger a characteristic transcriptomic signature, notably involving specific genes in defence phytohormone signalling.
Systematic large-scale microarray analyses reveal that the expression values of up to 30% of all genes probed on an array significantly correlate with degradation quality measures such as the 3′/5′-ratios of control genes [ 4, 21].
Microarray analyses reveal substantial differences in the global gene expression profiles induced by endoxifen at low concentrations (40 nM) when comparing MCF7 cells which express ERβ to those that do not.
Exposure of colon cancer cells to hypoxia results in altered expression of genes regulated by the AP-1 and NF- κB transcription factor families, with recognized functional consequences (Yao et al, 1994; Yao and O'Dwyer, 1995), and not unexpectedly, microarray analyses reveal the induction of multiple transcripts from various functional groups (Koong et al, 2000).
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Microarray analyses revealed that CBX6 governs a complex gene expression program.
Microarray analyses revealed that the expression of FN in irradiated cells was suppressed by combined use of RGD/P-AuNPs.
Microarray analyses revealed that nuclear export of a large number of intronless mRNAs is impaired in Drosophila-cultured cells RNAi knocked down for dU2AF50.
Microarray analyses revealed 37 possible OsbZIP23-specific target genes, which showed changes of reverse expression level in the overexpressor and mutant.
Microarray analyses revealed the upregulation of several transcription factor genes in barley and rice [41, 43], among which a bHLH transcription factor gene, IRO2, is of particular interest because of its pronounced transcriptional upregulation by Fe deficiency in shoots and roots of barley and rice [43].
Microarray analyses revealed that numerous developmental signaling pathways were affected by Hdac3-deficiency.
Indeed, microarray analyses revealed that several hundred genes were deregulated in tetR expressing U937 cells upon removal of tetracycline.
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