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We infected 3-week-old A129 mice with equal amounts of WT or NS1 mutant ZIKV (1 × 105 PFU) via the intraperitoneal route.
The resulting litters contained CRYAB+/-/HSPB2+/-, mice with equal numbers of non-transgenics and transgenics.
We subcutaneously injected SCID mice with equal numbers of MOCK or XRCC3 over-expressing MCF-7 cells (1.5×106 cells) in the absence of oestradiol treatment.
Each batch represents a pool of 3 independent mice with equal amount of cells from each mouse.
This experiment used 10 wildtype (C57Bl/6J) and 10 ephrin-A2 -/- knockout mice, with equal number of males and females.
Moreover, UV light exposure induces sleep in wild-type and melanopsin-deficient (Opn4 −/− ) mice with equal efficacy.
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Lymphopenic InsHA mice, coinjected with equal numbers of Clone 4 CD8+ and HNT CD4+ T cells, were treated every other day with 0,5 mg of the MR1 mAb.
A total of 727 mice, with about equal numbers of male and female mice, were used to determine the effect of gender on HCC formation (supplementary material Table S1A and B).
As expected, both cell lines demonstrated similar knockdown of endogenous mouse WDR5, with equal transcription of exogenous human WDR5 RNA.
Bone marrow cells from CD45.2+ IκBα-SR Tg mice were mixed with equal cell numbers from CD45.1+ wild-type mice and intravenously injected into lethally irradiated Rag1−/− mice.
For competitive repopulation assays, bone marrow cells (2 × 10) from in utero irradiated wild-type and KO (B6, CD45.2) mice were mixed with equal number of competitor bone marrow cells derived from congenic mice (B6, CD45.1) and transplanted into lethally irradiated recipients (CD45. 1/2, 6 8 weeks old).
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