Sentence examples for mice which reported from inspiring English sources

Our results correspond with investigations on mice, which reported decrease in level of RUNX2 after NaF exposure [ 5, 22].

The evaluation of EGPhE was based on a single study in mice, which reported a decreased number of live pups in the exposed group (Heindel et al. 1990).

To get insights on the functional impact of the selective SMAD3 epigenetic silencing in TAFs, we took advantage of a previous work on Smad3-null mice, which reported two skin fibroblast-associated altered functions in vivo: (i) increased ECM deposition in response to exogenous TGF-β1; and (ii) enhanced wound healing rate possibly due to enhanced contractility upon skin injury (36).

These findings were corroborated by in vivo imaging studies of AD transgenic (APP/PS1) mice, which reported a change in size of a subset of plaques [ 2] or a gradual growth of almost all plaques, which was faster in smaller plaques and younger animals [ 24].

However, this result was at odds with a previous study in mouse which reported that Spsb3 was highly expressed in muscles, rather than Spsb1 [28].

Our observations are consistent with previous studies in human alveolar epithelial cells and mouse macrophage cells which reported that PEGylated NPs formed smaller intracellular agglomerates than did bare AuNPs [22, 39] and experienced decreased cellular uptake [40].

The most similar protein with known function is ubiqulin 1 (NCBI Protein Accession Nos. NP_444295. 1 for humans and NP_689420. 1 for mice), which is reported to be associated with ubiquitin ligases and proteasomes (Ko et al. [2004]).

The genetic interaction observed here in the context of filopodia formation is consistent with genetic interactions observed in Mena−/− profilin-1−/− mutant mice which were reported to display defects in neural tube closure [35].

Interestingly, 25% of Emilin1−/− mice die prematurely between 14 and 18 months due to unclear causes when compared with Emilin1+/+ mice, which are reported to have a longevity of 25 months (Yuan et al., 2009).

Transgenic mice hemizygously expressing mutated human α-synuclein (A53T M83+/−: Gfap-luc mice) which were reported to show no protein aggregation or clinical disease until at least 629 days of age.

APP (Tg2576 mice; APP23 mice; APP23: Gfap-luc mice; APPSwe/PSENΔE9 mice; APPPS1 mice), tau (B6/P301L mice; P301S mice; P301S PS19 mice), or α-synuclein (homozygous A53T M83+/+ mice, Fig.  2) Transgenic mice hemizygously expressing mutated human α-synuclein (A53T M83+/−: Gfap -luc mice) which were reported to show no protein aggregation or clinical disease until at least 629 days of age.