Sentence examples for mice which died from inspiring English sources

We generated HAI-1-deficient mice, which died in utero due to placental defects.

Complete absence of p27Kip1 had a major effect in Cdk4(+/R) or Cdk4(R/R) mice, which died within the first 7 months of life.

Unlike the DLC1−/− mice, which died in embryonic stage, DLC2−/− mice could survive to adulthood.

A protective role for adaptive T cell mediated immunity against LVS infection was demonstrated in genetically immunodeficient mice, which died of overt infection one month after intradermal innoculation [13].

RAG2−/− mice infected with PbAluc did not develop ECM contrary to wild-type (WT) C57BL/6J mice which died with neurological signs 6 to 12 days post-infection (Figure 1A).

Unlike Taspase1−/− mice, which died prematurely, MLL nc/nc mice were viable and fertile.

The first evidence demonstrating a scaffolding role of RIPK1 in preventing necroptosis came from studies using transgenic mice showing that loss of RIPK3 protects RIPK1/caspase-8 KO mice, which die shortly after birth16,39,40.

Unlike Ripk1 mice, which die shortly after birth, Ripk1(D138N/D138N) mice are viable.

In contrast to GRK2-deficient mice, which die in utero (Jaber, M., Koch, W. J., Rockman, H., Smith, B., Bond, R. A., Sulik, K. K., Ross, J. JR., Lefkowitz, R. J. Caron, M. G., and Giros, B. (1996) Proc.

This experimental cerebral malaria (ECM) model is characterized by the intravascular accumulation of immune cells in the brains of mice which die during the first 10 days of infection with signs of neurological involvement [20] and evidence of circulatory shock [21].

NL2 KO mice exhibit irregular breathing patterns much like NL1/2/3 triple-KO mice, which die within 24 h after birth because of breathing failure [ 48].