Sentence examples for mice were unresponsive from inspiring English sources

Thymocytes from SH2D2A+/+ and SH2D2A−/− mice were unresponsive to 10 ng/ml CXCL12 (data not shown), whereas 100 and 500 ng/ml of CXCL12 induced significantly greater migration of SH2D2A+/+ than SH2D2A−/− thymocytes (Fig. 5B).

These mice were unresponsive to treatment with regard to IgG and IgE secretion, cellular infiltration and epithelial hyperplasia.

When the mice were unresponsive to toe-pinch, they were intubated and ventilated with a rodent respirator and were placed on a heating pad.

The gustatory nerves in these mice are unresponsive to taste stimuli.

These mice are unresponsive to bacterial lipopolysaccharide (LPS), an integral component of the outer membranes of Gram-negative bacteria that can cause endotoxin shock.

The anti-inflammatory and proresolving effects of C15 are mediated by ChemR23 because ChemR23−/− cells and mice are unresponsive to the peptide, whereas neutralization of endogenous chemerin species results in exacerbation of peritonitis [16,49].

Based on these findings, it would appear the DBA/2 mouse was unresponsive to CSC dermal tumor promotion.

These mice were then challenged with either one of two different tumors that were demonstrated to be responsive to αOX40-mediated tumor immunity in young mice, but were unresponsive in 12-month-old as well as elderly 20-month-old mice.

Genetically engineered, TLR4-deficient mice (C57BL/6 TLR4-/- C57BL/6 TLR4-/-sive to inhaled lipopolysaccharide, except for minimal increases in some inflammatory cytokines.

Our results from the previous experiments showed that the SCPP responses of juvenile B6 mice were generally unresponsive to modifications of the social conditioning environment.

For the analysis of SCPP (Experiment 2), 19% of A mice and 28% of BALB mice were similarly unresponsive, expressing deviant place-preference scores and exploratory behavior (>3.5 SD and >2 SD, respectively, as reported above).