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Quantitative results from all the three mice were summarized in Figure 5(c), showing that NSC vectors displayed induced luciferase gene expressions in tumor sites by averagely 30 folds higher than those in sham sites.
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The percent parent compound 11C-dLop at different time points p.i. in mice are summarized in Table 1.
Biodistribution data of [64Cu]NOTA-pentixather at 1.5 h p.i. (n = 5, black bars) and 24 h p.i. (n = 6, grey bars) in Daudi-lymphoma-bearing SCID mice are summarized in Fig. 3.
Hematology and clinico-pathologic findings in S. Typhimurium-infected Sv129S6 mice are summarized in Figure 1 (16-week study), and Tables 1 and 2 (10-week study).
The combined data from all of the treated mice is summarized in Figure 2B and confirms that 120G8 significantly reduces only pDC and not cDC subsets.
The results of the tissue distribution studies of the two radiotracers in mice are summarized in Tables 1 and 2. The highest initial uptake (percentage of injected doses per gram of tissue: %ID/g) of [11C]A-582941 was found in the lungs followed by the kidneys, pancreas, heart, liver, small intestines, spleen, brain and muscle (Table 1).
The incidence, median survival, range of survival and presence of ascites for these groups of mice are summarized in Table 3 with the Kaplan-Meier survival plots depicted in Figure 2. When compiled, these data indicate that loss of p53 is necessary for tumor formation and that Brca1 deficiency accelerates tumor initiation and/or progression.
The timelines for treatment of donor mice are summarized in Figure 8a.
The physiological and biochemical parameters of the mice are summarized in Table 1.
The acute toxicity of aqueous Aquilaria crassna leaf extract in mice is summarized in Table 3.
The strategy to generate pBmpr1aKO mice was summarized in supplementary material Fig. S1.
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