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Post-hoc Neuman-Keuls testing indicated that both the wild-type controls and treated Tg mice were significant different from the control Tg mice (p<0.001).
These differences in tumor incidence between the Wip1+/+ and Wip1−/− mice were significant (P = 0.016).
The differences between individual mice were significant (p = 0.01) and highly significant between the counting methods (p < 0.001).
Besides, hPEDF levels in serum and ascites of rAAV2-hPEDF-treated mice were significant higher than those in rAAV2-null or normal saline (NS) groups.
Differences in viral and titers between naïve and age-matched vaccinated mice were significant for the following group: young AdC68NP/A/X31: P = 0.048).
As shown in Fig. 2 (panels c and d) the differences in tumor size between asPR-treated animals and vehicle-treated or scPR-treated mice were significant.
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The body weight of ob/ob mice was significant higher than that of WT as indicated by previous studies (Fig. 2A).
The difference in response to dendrotoxin-K in wild-type and mceph/mceph mice was significant, P = 0.02 (Fig 4C).
A reduction of the CD4+ T-cell population in C3.Dll1tm1Gos/+ mice was significant only in females (Table 1).
This 2°C difference between WT and N2KO mice is significant between the two genotypes (*p<0.001, T value = −6.40, df = 2).
Quantitative analysis performed on paraffin-embedded sections showed that at five months of age, the loss of photoreceptors in mutant mice was significant, with Guca1a+/COD3 mice retaining 69% of cells in the ONL compared with wild-type littermates (Figure 8d).
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