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Uteri from STS-135 mission mice were preserved in RNA later for 10 months at −80°C, prior to processing in RIPA as described above.
Both hypercholanemia and normocholanemia in Slc10a1 −/− mice were preserved during the backcross to C57Bl/6J (from 50% genetically similar to 129/SvEv to >98% similar to C57Bl/6J), excluding effects of different mouse strain backgrounds.
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Histological data presented in this study show that enterocytes architecture in the colon of infected mice was preserved in the presence of Sb during ST infection.
Remarkably, whereas the small intestines from HLA-DR3.IFN-γ+/+ transgenic mice displayed significant pathological changes during TSS, the architecture of small intestines in HLA-DR3.IFN-γ−/− transgenic mice was preserved.
We found that spontaneous activity of the rHIgM12-treated mice was preserved compared to controls.
In our VILI experiments, lung function in APC-treated mice was preserved compared with mice receiving saline, which could be interpreted as protection against pulmonary edema formation by APC.
There was no alteration of vascular relaxation in response to NO donor SNP in both diabetic mice and transgenic mice with calpastatin over-expression as compared to non-diabetic and wild-type mice, respectively, suggesting that the function of smooth muscle relaxation in aortic rings of diabetic mice is preserved.
The difference in mean arterial blood pressure between wild type and SPAK243A/243A knock-in mice was preserved over the course of both day and night, with higher blood pressure measured at night when animals are more active (Fig 2A and B).
The mice were sacrificed by CO2 inhalation and were dissected on site and organ tissues from each mouse were prepared and later each mouse was preserved in absolute Ethanol for morphological analysis and future analysis.
However, while in chimaeric DG-null mice the NMJs were disrupted, in MCK-DG-null mice they were preserved.
While dihydroxyacetone-P (DHAP) and glycerol-3-P levels were decreased in the fasted state of the FAAH−/− mice, they were preserved in the fed state, consistent with a compensating contribution from a decrease in fed aldolase B acetylation in FAAH−/− mice.
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