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In contrast, transgenic mice were, on average, unable to improve their performance in finding the hidden platform over trials.
Second, uko/mdx mice were on the BL10 background [11].
All mice were on the FVB/N strain background.
Pdx1-tTA knock-in mice were on an ICR genetic background [13].
Thrombospondin-1−/− mice were on a C57/Bl6 background as described previously [53].
All mice were on a C57BL/6 background and genotypes were determined by PCR.
However, uko/3cv mice were on a mixed genetic background of BL10 and C57Bl/6.
The mice were on average 20 to 25 g (5 8 weeks old) at the start of the experiments.
Nine-week-old Myd88−/− (MyD88 KO) mice [7] and control wildtype mice were on a C57BL/6 background.
All mice were on a pure C57BL/6J background, and have been backcrossed to C57BL/6J for 13 generations.
NZB/BlNJ mice were on average the least affected by the treatments and as such the most distant from the others.
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