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By the fourteenth week of age, all mice were identifiable by histology (5 of 5), and the afflicted regions were larger and mostly devoid of neurons.
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Each mouse was identifiable with a numbered tag.
The originally mutated mouse was identifiable because the dystrophin gene is on the X-chromosome and the original male could be assessed in the hemizygous state.
At the time of eye opening (postnatal day 12), Rab18 −/− mice were readily identifiable by the presence, with complete penetrance, of dense nuclear cataracts (Fig. 1G), which were absent in the eyes of control mice (Fig. 1F), and atonic pupils, recapitulating the characteristic ocular phenotypes that are associated with WARBM.
Development of the palate begins around week 6 of gestation in humans (embryonic day E12 in mice) and is identifiable by the appearance of palatal primordia at the lateral edges of the maxillary prominences.
Osteoarticular infections were identifiable and consistent in mice infected with a range of infectious inoculum doses (Fig. 1; supplementary material Fig. S1).
The shots were identifiable.
Triggers were identifiable in 71 (78.8%) patients.
No seasonal cryptosporidiosis patterns were identifiable.
Some OTUs were only identifiable to kingdom while others were identifiable to genus.
Detailed renal analysis at 4 weeks of age using electron microscopic analysis (Fig. 2) showed that the ultrastructure of kidney was altered in POKO mice compared with ob/ob and WT mice, with specific abnormal architectures being identifiable.
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