Sentence examples for mice were glucose from inspiring English sources

Intraperitoneal glucose tolerance tests, performed in non-obese 5-week-old mice, showed that the STAT3-insKO mice were glucose intolerant.

However, PARP-2 deleted mice were glucose intolerant and exhibited pancreatic dysfunction, implying that these results may interfere with other beneficial consequences of PARP inhibition, and hence warrant further investigation on the safe clinical use of these inhibitors [5].

Because TP53INP1-deficient mice were glucose intolerant, and since TP53INP1 transcripts were significantly increased in islets isolated from HFD-fed mice (Fig 3E), we next hypothesized that defects in β-cell function or plasticity could occur in TP53INP1 knockout mice.

First, we confirmed that PPARβ−/− mice were glucose intolerant at 4 and 18 months and found that this alteration was maintained, but not exacerbated, as they age (Supplemental Figure 1A).

Three weeks after administration of Tam, nearly all of the β-cells had deleted the wild-type eIF2α cDNA, although there was no significant change in islet mass or insulin content and the mice were glucose intolerant [76].

BAC/+ mice were glucose intolerant and BAC/MIP mice displayed the most severe glucose intolerance of the three groups, even with the attenuated glucose injection, confirming our earlier results (Supplementary Material, Fig. S1A and B).

Such mice were glucose-intolerant, but their pancreatic phenotype was not described.

ASM−/− mice are glucose intolerant.

These results demonstrate that the AS160 knockin mice are glucose intolerant.

Taken together, these findings suggested that the Aldh1b1tm1lacZ null mice are glucose intolerant due to islet defects in glucose-stimulated insulin secretion (GSIS).

The E2 increase of GLUT4 expression is likely mediated through ER α since ER αKO mice are glucose intolerant and insulin resistant [ 28].