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The IGF-I receptor knockdown mice were fertile, slightly smaller and resisted oxidative stressors more effectively when compared with normal littermate mice.
The homozygous Rag2-R229Q knock-in (heRag2ter Rag2 KI) mice were fertile and survived into adulthood with no obvious development defects, compared to wild-type strains (hereafter Rag2 WT; data not shown).
In OPN mutant mice, embryogenesis occurred normally, and mice were fertile.
CD177−/− mice were fertile and litters were born at a normal Mendelian ratio without any discernible phenotype (data not shown).
CXCL14−/− male mice were fertile.
Both male and female mice were fertile.
Similar(24)
Previously we demonstrated anthrax toxin receptor 2 knockout (Antxr2−/−) mice are fertile but fail to deliver their pups at term.
Drf1−/−RhoB−/− mice are fertile and develop normally.
Heterozygous mice are fertile and display no obvious pathology.
In agreement with this notion, TRPM8 null mice are fertile [49].
Homozygous NOS2 mutant mice are fertile, and display no developmental defects or abnormalities of blood composition.
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