Sentence examples for mice were driven from inspiring English sources

This is a crude model of body temperature oscillations in vivo, however when cultured under these conditions paired cultures from single mice were driven into antiphase.

33 We reasoned that if the differences in seizure progression observed between the tPA −/− and Nsp −/− mice were driven by differences in BBB integrity, then these phenotypic differences in seizure progression would be absent in an ex vivo preparation where the BBB plays no role in regulation of the parenchymal extracellular environment.

Augmented musclin production in TG mice is driven by a molecular cascade resulting in enhanced acetylation and nuclear exclusion of the transcription factor forkhead box O1 (FOXO1) – an inhibitor of transcription of the musclin encoding gene.

The expression of h-S100A7 transgene in transgenic mice is driven by the cytomegalovirus immediate-early enhancer linked to the chicken beta-actin promoter.

Ongoing insulitis, as a hallmark of T1D in NOD mice, is driven in part by the persistence of activated, autoreactive T and B lymphocytes.

The expression of h-S100A7 transgene in these transgenic mice is driven by the cytomegalovirus immediate-early enhancer linked to the chicken β-actin promoter.

We propose that the affiliative social interactions of young mice are driven by a state of reward and that differences in the nature of these interactions can be moderated by genetic factors during a relatively narrow window of adolescent development.

Since the central clock in Per1−/− Per2m/m mice is driven by environmental light in LD cycles but is not functional in DD cycles [52], these mice could maintain a circadian rhythm of urinary catecholamine levels in LD cycles but not in DD cycles.

Co-immunofluorescence studies revealed that the PPARβ/δ transgene was not found in either endothelial cells or dermal dendritic cells (fig. S3), further confirming that the skin disease in PPARβ/δ transgenic mice is driven by expression of the transgene in suprabasal epidermal keratinocytes, but not other cell types.

Expression of alpha-synuclein in M20- and M83-mice are driven from the prion promoter, while expression of human alpha-synuclein in Tg SNCA 1Nbm/J mice is driven from a P1 artificial chromosome, which contains the human promoter for alpha-synuclein.

The aggressive progression of mammary lesions in BALB-neuT mice is driven by the activated rat HER-2/neu oncogene [ 28].